Cases reported "Duodenal Obstruction"

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1/6. Annular duodenal stricture due to Brunner's gland hyperplasia.

    A patient with obstructive Brunner's gland hyperplasia presenting as an annular duodenal stricture is reported. Surgical biopsy was required to obtain a tissue specific diagnosis and obstruction was relieved by performing a Roux-en-Y duodenojejunostomy. Brunner's gland hyperplasia poses a diagnostic challenge. Conservative management is usually adequate after a histological diagnosis has been firmly established.
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2/6. Villoglandular adenoma of the duodenum.

    Two cases of villoglandular adenoma of the duodenum presenting with features of peptic ulcer are reported. At surgery both patients had intussusception of the duodenum. One of them had carcinoma in situ. The literature is briefly reviewed, and the importance of keeping in mind this rather rare condition, even in areas where duodenal ulcer is widely prevalent, is emphasized.
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3/6. Brunner's gland adenomas: clinical presentation and surgical management.

    Tumors of the Brunner's glands are rare, and the etiology remains obscure. Bleeding is the most common presenting symptom and may be occult or exsanguinating. Gastric outlet or duodenal obstruction may also occur. Often there is a history of preexisting nonspecific upper gastrointestinal symptoms, or the adenoma may be an incidental postmortem finding. Although contrast studies usually suggest the diagnosis confirmation requires endoscopy or operation. Resection is the preferred therapy, but bypass of the lesion has been done becaue the adenomas are not considered premalignant. Gastroduodenoscopy may facilitate definitive management. Our experience in managing three patients with Brunner's gland adenomas, including a patient with life-threatening upper gastrointestinal hemorrhage from an ulcerated Brunner's gland tumor, is cited.
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4/6. Endoscopic removal of a large, obstructing and bleeding duodenal Brunner's gland adenoma.

    About 10-30% of benign duodenal tumors are Brunner's gland tumors. These are almost always benign. Usually they present as incidental findings in the form of small polypoid lesions at barium examination or at panendoscopy. We present a case with a large obstructing and bleeding Brunner's gland adenoma located in the duodenal bulb which was removed by endoscopic polypectomy.
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5/6. hyperplasia of Brunner's glands of the duodenum.

    hyperplasia of Brunner's glands is a rare phenomenon. It may cause obstructive symptoms, anemia or the patient may be totally asymptomatic. The diagnosis can be confirmed with endoscopic examination and upper gastrointestinal series. The surgical treatment or hyperplasia of the Brunner's glands should be conservative since the lesion is not premalignant. If complications do occur, local excision is the treatment of choice. Two patients are reported who had Brunner gland hyperplasia as an incidental finding at exploration for pancreatic pseudocyst, and a brief review of the literature is made.
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6/6. The interpretation and management of duodenal lesions other than benign ulcer.

    Five patients, each with a unique disease, illustrate some of the unusual subtleties of interpretation, the challenges of operative approach, the avoidance of pitfalls and the management of complications in local duodenal surgical procedures. A Brunner's gland polyp was resected reversing prior pancreatitis and cardiac alterations. A rare ampullary gangliocytic paraganglioma, hemorrhaging, was managed by duct isolations, resection and papilloplasty. A perforated malignant bleeding ulcer with eventual common bile duct obstruction and mimicking a benign ulcer, was approached with a variety of effective palliative procedures. A complicated duodenoileocolic cutaneous fistula in a patient with a background of granulomatous disease was resected. Finally, an obstructing duodenal stricture, attributable to progressive pancreatitis and presenting two years after bypass for jaundice, was managed by gastrojejunostomy and vagotomy, later converted to a Roux-en-Y preparation because of poor emptying and reflux bile gastritis.
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