Could someone help me about having a diabetes mellitus?

---

Today i found out that ants were feeding on my urine! I remembered that if ants feed at your urine it's a sign of diabetes mellitus. and today i have a frequent urination more than yesterday.. I'm also guilty that I had ate and drank too much sugar today, i'm just wondering if im having a diabetes or it's just my glucose level is high today? can someone please help me cause I know diabetes is one of the main genetic disease my family have.
These are indeed signs of diabetes. Even if you eat an obscene amount of sugar, your system should quickly and efficiently bring blood sugar level down to proper levels, somewhere below 100. Since others in your family have diabetes, ask someone to test your blood. If it is above 126 you almost certainly are diabetic. If it is above 100 and below 126 you may be borderline and easily controlled through diet and exercise and nothing more.  (+ info)

Is Charcot Marie Tooth Disease associated with Diabetes Mellitus?

---

I was diagnosed with Type II Diabetes about 3 years ago. It has progerssively gotten worse. I was diagnosed with Diabetic Polyneuropathy last year as well. I was always described as a "clumbsy" child. My mother said I was the only person she knew that could fall down without taking a step.

My neuro says I have a gait disturbance but has done nothing as far as telling my why.

I had some foot pain the last three days and had an x-ray done. My Internist told me I have arthritis and bone spurs but it could be Charcot Marie Tooth Disease.

Is there any information out there linking Charcot Marie Tooth Disease and Diabetes?
Are you confused on the name? Charcot-Marie-Tooth (CMT) disease is named for the physicians who identified the disease, Charcot, Marie, and Tooth. It is not the same as Charcot's foot disease, a neuropathic joint disease that is a common complication of diabetes mellitus. http://www.neurologychannel.com/charcot/
For more info on Charcot's foot disease, look here: http://www.diabetes.usyd.edu.au/foot/Charcot1.html  (+ info)

Would a person with diabetes mellitus be likely to require more insulin or more sugar after strenuous exercise?

---

I really dont understand this question. I have looked all over the internet and it doesnt really say. Can someboy PLEASE help me
No. Exercise lowers blood sugar so you may need an extra snack.  (+ info)

What complications or disease can arise from Diabetes Mellitus Type 1, child onset?

---

and how does diabetes cause these complications or diseases?
kidney problems,blindness,poor circulation,poor wound healing,amputation of legs, arms,feet and heart problems.  (+ info)

Is it right to consume honey while I have diabetes mellitus type 2?

---

sugar is strictly forbidden for diabetes patients but some alternative medicine mix honey to their medicine
Count your carbs as usual.  (+ info)

Why is diabetes mellitus sometimes called "starvation in the midst of plenty"?

---

Thank you so much in advance!
The simplest sugar, Glucose, is used as fuel for your body. You NEED some glucose in your blood to fuel the cells of your body, especially the muscles. Without fuel, the cells will fail to function and eventually die.

In order for the glucose in the blood to pass from the blood into the cells, a hormone called INSULIN is needed. Insulin is a CATALYST -- it help move the sugar from the blood into the cells.

The purpose of digestion is to break down the food we eat into glucose, water, vitamins and minerals, and carbon dioxide. The process of digestion puts glucose into the blood.

When a person is diabetic, one of two things happens:
1) The body does not produce enough insulin.
2) The body cannot use the insulin it produces.

In either case, the result is the same -- the glucose builds up in the blood, but never passes into the cells.

The cells, not being able to get the glucose they need, give off chemical signals saying "WE'RE HUNGRY!" Even though there is plenty of glucose (even too much) in the blood stream, the celsl think they are starving!

So diabetics are often very hungry -- but eating does not good. The cells don't get their glucose, and continue to send out the "starving" signals.

What is worse, the amount of glucose in the blood goes high and higher. This does all kinds of damage to the internal organs, eyes, brains, and nerves. It damages the kidneys, which makes the blood pressure go up, which causes heart attacks and strokes. Since the cells don't get the fuel they need, healing is much slower than normal. Infections often set in, and it is not uncommon for gangrene to result, causing amputation of fingers, toes, hand, feet, and legs.

If the diabetes is left uncontrolled, the body will go into "starvation mode". The body will actually start to digest itself! But in starvation mode, the body actually starts to digest muscles. This produces a great deal of toxins in the system called KETONES. Too many ketones in the blood (called KETOSIS) will raise the blood acidity level, causing KETOACIDOSIS.

If the patient continues untreated, ketoacidosis is fatal. The patient goes into a coma, and the acidic blood levels eventually cause a heart attack and/or irreversible kidney failure.

Many patients (like myself) who have had an attack of ketoacidosis find that their internal organs, especially the kidneys, are permanently damaged. Damaged kidneys will eventually require dialysis or (if you are lucky) a transplant.  (+ info)

What group of people are more likely to develop non-insulin dependent Diabetes mellitus II?

---

If there's type 2 in your family, you are at risk for developing it. Type 2 is more hereditary than type 1.

If you are predisposed, but pay good attention to your health (eat right, exercise, etc) you are less likely to develop type 2 than a brother/sister who may also be predisposed but eats junk and never gets of the couch.

People are surprised at the growing number of type 2 diagnoses, and puzzled by its diagnosis in kids. Fact is, it's always been around, Americans just got wayyy lazier. We live in a world of fast food, remote control and video games. Pre-disposition is there or it's not, but it doesn't have to evolve into the disease.

That's not to say you WONT get type 2 even if you do everything right. It's just far less likely.  (+ info)

Is too much sugar directly responsible for diabetes mellitus type 2?

---

To avoid sugar after the onset of diabetes mellitus is well known... but could avoiding sugar before the onset of the disease reduce the occurence of the disease... if so whats the pathophysiology....
No, too much sugar is NOT responsible for diabetes. And after the onset of type 2 it is important to reduce carbohydrates, sugar just happens to be one. It is just as important to watch how much rice or potatoes or white bread or milk is consumed as it is sugar.

Also "Fully" needs to work on her facts and stop going to "that place where all the people go that know nothing about diabetes" because her "facts" are wrong! She may be a type 1 diabetic but that does not make her an expert on diabetes.

Type 2 is a type of diabetes mellitus and is not brought on by bad diet, obesity, lifestyle etc, but is a genetic or hereditary disease.  (+ info)

What urinalysis tests can diagnose diabetes (mellitus and insipidus) and jaundice?

---

Urinalysis can detect Diabetes Mellitus using the urine strip. Urine chemical examination is a screening test for Diabetes and Proteinuria. Not jaundice.

Jaundice may be caused by Hepatis virus or Liver disease.

You can find out the reasons of jaundice with laboratory exams such as Hepatitis B surface antigen, SGPT, Bilirubin.  (+ info)

Can someone explain the pathophysiology of how diabetes mellitus causes lactic acidosis?

---

Lactic acidosis is the most common cause of metabolic acidosis in hospitalized patients. It is associated with an elevated anion gap and a plasma lactate concentration above 4 meq/L. Impaired tissue oxygenation, leading to increased anaerobic metabolism, is usually responsible for the rise in lactate production. (See "Approach to the adult with metabolic acidosis").

The pathophysiology and causes of lactic acidosis will be reviewed here. The possible role of bicarbonate therapy in such patients is discussed separately. (See "Bicarbonate therapy in lactic acidosis").

PATHOPHYSIOLOGY — A review of the biochemistry of lactate generation and metabolism is important in understanding the pathogenesis of lactic acidosis. Both overproduction and underuse of lactate appear to be operative in most patients.

Lactic acid is derived from the metabolism of pyruvic acid; this reaction is catalyzed by lactate dehydrogenase and involves the conversion of NADH into NAD+ (reduced and oxidized nicotine adenine dinucleotide, respectively). Normal subjects produce 15 to 20 mmol/kg of lactic acid per day, most of which is generated from glucose via the glycolytic pathway or from the deamination of alanine [1,2].

Lactic acid is rapidly buffered, in part by extracellular bicarbonate, resulting in the generation of lactate. In the liver and, to a lesser degree, in the kidney, lactate is metabolized back to pyruvate, which is then converted into either carbon dioxide and water (80 percent, catalyzed in part by pyruvate dehydrogenase) or glucose (20 percent, catalyzed in part by pyruvate carboxylase). Both of these processes result in the regeneration of the bicarbonate lost in the initial buffering of lactic acid.

And now you know the rest of the story !  (+ info)

---

---

• Home
• Our method
• Usage examples
• Index

• Statistics
• Contact

---