is there a cure for rhuematic chorea?

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I have had rhematic chorea when I was 13. It has caused me have rheumatic heart disease. Is there a permanent cure for this, apart from valve replacement?
U R SUFFERING FROM WHAT IS CALLED A RHEUMATIC HEART DISEASE. U MUST HAVE TAKEN PENICILLIN PROPHLAXIS FOR IT I AM SURE .I DO NOT KNOW UR AGE BUT USUALLY THE PROPHLAXIS IS TAKEN FOR 5YRS FROM THE TIME U R DIAGNOSED OR TILL U TURN 21 WHICH EVER COMES FIRST. THE CHANCES R SAID 2 B VERY LITTLE AFTER 30 OF U GETTING THE SYPMTPS BUT U MUST TAKE CARE WHENEVER U GET A RESPIRATORY INF EG SORETHROAT ETC  (+ info)

Is there anyone else out there who has a family member with Huntington's Chorea Disease?

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Is there anyone else out there who has a family member with Huntington's Chorea Disease? If so, what state do you live in?
Hi. Yes I have HD in my family. I currently live in Cali but I'm originally from Syracuse, NY.  (+ info)

Does this mean I have huntingtons? Does huntingtons chorea mean the disease to?

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My hands and legs r always shaking. My hands shake so bad now i can't even do normal things like write barely or put on jewelry. I'm in my early 20s. Does this sound like huntingtons disease or chorea? If it's huntingtons chorea does it mean i have huntingtons disease.?
Chorea is a symptom; jerky, sporadic movements. Huntingtons is a genetic disease, if no one in your family has it then it's very unlikely. See a Dr for a blood test if you're still concerned. There are many reasons for tremors.

Huntingtons chorea is the old name for huntingtons disease.  (+ info)

What causes Chorea in Rheumatic Fever?

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im havin a hard time understanding how rheumatic fever causes dance like movements? what's the cause of sydenham's chorea in rheumatic fever?
experts suggest that Sydenham's chorea appears to result from such an autoimmune response. Evidence indicates that certain streptococcal proteins or antigens (streptococcal M proteins) induce the body's production of antibodies (i.e., antineuronal antibodies) that "crossreact" against the body's own cells in certain regions of the brain. (Group A streptococcal M protein has been shown to contain some of the same amino acid sequences as within certain human tissues.) Furthermore, some researchers have reported detecting certain antibodies (e.g., immunoglobulin G [IgG] antibodies) in children with Sydenham's chorea that interacted with certain cellular proteins (i.e., neuronal antigens) in the basal ganglia, such as the caudate nuclei and subthalamic nucleus. The basal ganglia are paired nerve cell clusters deep within the brain that play an essential role in initiating and regulating movement.
pls read the rest of the article at http://www.wemove.org/syd/syd_cau.html  (+ info)

can i get a driving license even if i have a neurological condition it is called moya moya i also have chorea?

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The only people who can answer this question are the DVLA - why don't you ask them, instead of a load of total strangers on here, who don't know your medical history or the DVLA regulations?  (+ info)

Are there any recent advancements for huntington's chorea/disease?

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Please provide links.
try this site:

http://hddrugworks.org/

and this:(it is about Parkinsons but mentions Huntingtons disease. Taken from: http://www.mdidea.com/products/herbextract/coenzyme/data.html

Coenzyme Q10 and Parkinson's disease:

Low levels of natural coenzyme Q-10 have been observed in individuals with muscle-wasting diseases (conditions that result in decreased muscle size and efficiency). Therefore, coenzyme Q-10 is being studied as a possible treatment for conditions, such as Parkinson disease (PD), that affect muscle function. Coenzyme Q-10 possible energy-enhancing effects may prevent the deterioration of muscle activity. Additionally, in animal and human studies, increasing amounts of coenzyme Q-10 also seemed to increase levels of a neurotransmitter known as dopamine. Neurotransmitters are chemicals that carry messages from nerve cells to other cells. Individuals with PD generally have low dopamine levels, so raising dopamine may relieve their PD symptoms. Additionally, coenzyme Q-10 may reduce other factors, such as inflammation and damage by oxygen free radicals, that may cause or worsen PD. Studies are less conclusive, however, for coenzyme Q-10 possible effectiveness for other muscle-wasting conditions such as HUNTINGTON DISEASE and muscular dystrophy. While these and similar conditions may have a connection to low coenzyme Q-10 levels, it is not known if the lowered levels of coenzyme Q-10 contribute to the conditions or result from them. Much more research is needed in these areas.
Results of the first placebo-controlled, multi-center clinical trial of the compound coenzyme Q10 suggest that it can slow down disease progression in patients with early-stage Parkinson's disease. While the findings must be confirmed in a larger study, they provide hope that coenzyme Q10 may ultimately provide a new way of treating Parkinson's disease.
The phase II study, led by Clifford Shults, M.D., of the University of California, San Diego (UCSD) School of Medicine, looked at a total of 80 Parkinson's disease patients at 10 centers across the U.S. to determine whether coenzyme Q10 is safe and whether it can slow down the rate of functional decline. The study was funded by the National Institute of Neurological Disorders and Stroke (NINDS) and appears in the October 15,

2002 issue of the Archives of Neurology.
"This trial suggests that coenzyme Q10 can slow the rate of deterioration in Parkinson's disease," says Dr. Shults. "However, before the compound is used widely, the results need to be confirmed in a larger group of patients."
The researchers believe that coenzyme Q10 works by improving the function of mitochondria, the 'powerhouses' which produce energy in cells. Coenzyme Q10 is an important link in the chain of chemical reactions which produces this energy. It's also a potent antioxidant - a chemical that "mops up" harmful free radicals generated during normal metabolism. Previous studies carried out by Dr. Shults, Richard Haas, M.D., of UCSD and Flint Beal, M.D., of Cornell University have shown that coenzyme Q10 levels in mitochondria in Parkinson's disease patients are reduced and that mitochondrial function in these patients is impaired.

Animal studies have shown that coenzyme Q10 can protect the area of the brain which is damaged in Parkinson's disease. Dr. Shults and his colleagues also conducted a pilot study with Parkinson's disease patients which showed that consumption of up to 800 mg/day of coenzyme Q10 was well tolerated and significantly increased the level of coenzyme Q10 in the bloodstream.
However, in terms of Parkinson's disease, prevention is clearly the best option. The single best thing one can do is avoid pesticide and insecticide exposure.
Go Top  (+ info)

Hey i was wondering who pays for the tests of the disorder "Huntingtons disease or hungtingtons chorea" ?

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??? I'm not sure I understand your question. Why would anyone other than the patient or their insurance company pay for any diagnostic test?  (+ info)

hi does any body know anything about the genectic disease huntingtons chorea?

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any info will be helpfull thanks
autosomal dominant ,50-50 chance of inheritance ,clinical picture manifests around 40 years old ,its genetically determined to damage the neural tissue ,  (+ info)

What can stimulate Huntington chorea symptoms?

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Background: Huntington disease (HD), also known as Huntington chorea (HC), is an inherited disease characterized by choreiform movements and progressive dementia.

In adults, HD most often causes involuntary movements, but rigidity can also be a feature of the disease.

The initial diagnosis is rarely established in the emergency department, but patients with established disease may present to the ED because of worsening symptoms.


Pathophysiology: HD is inherited as an autosomal dominant disorder with complete penetrance. An HD gene has been identified with an abnormal protein product (huntingtin) that can be identified in the brain. The link between this protein and the selective loss of neuronal groups in the CNS remains to be established. HD has now been identified genetically as a trinucleotide CAG-repeat mutation on chromosome 4. The CAG repeat length may be important in determining the age of onset and the rate of disease progression.


Frequency:


In the US: Prevalence of HD in the United States is 5.15 cases per 100,000 persons.
Internationally: HD is encountered throughout the world; however, localized geographic clusters of disease exist. Countries that have been settled by western Europeans have an incidence of the disease similar to the incidence in the United States.
Mortality/Morbidity: HD is a progressive neurological disorder usually leading to death 15-20 years after onset of neurological or psychological impairment.

Race: HD is found in all ethnic groups.

Sex: Males and females are diagnosed in equal numbers.

Age:

Symptoms arising from a typical presentation of HD usually do not develop until a person is aged 35 years or older. By the time of diagnosis, many patients already have had children and have passed the gene to another generation.
As many as 10% of patients with HD have a juvenile form of the disease in which the onset of symptoms may occur when the patient is younger than 20 years.
Muscular rigidity is more common with juvenile-onset illness.  (+ info)

is there any cure for huntington chorea disease ?

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i kw that there is not a complete cure for this disease but to some extent in countries like usa does have somewht cure ....can u plzz help and tell me some cures for this disease...!!
As I understand it there is no cure at all for Huntington's, but there is some medicine that helps ease symptoms.  (+ info)

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