Cases reported "Zygomatic Fractures"

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1/15. Miniplate osteosynthesis and cellular phone create disturbance of infraorbital nerve.

    A 37-year-old man with a zygomatic fracture underwent surgical treatment with reduction of the fracture and osteosynthesis with a miniplate on the infraorbital rim. Postoperatively, he had numbness in the distribution area of the infraorbital nerve, but he also suffered from dysesthesia in the same area during periods when he was using his hand-held mobile phone. After surgical removal of the osteosynthesis plate, the dysesthesia associated with his mobile phone was no longer present. The plate was examined in a setup where we measured the electric current that developed on the surface of the plate under the influence of the magnetic field between the phone antenna and the metal plate. The highest currents measured on the actual plate were 141 mV in air, and 21 mV in saline. These findings indicate that there might have been a correlation between the presence of the miniplate close to the infraorbital nerve, and the dysesthesia experienced by the patient, under the influence of the energy emitted from the cellular phone.
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2/15. C-shape extended transconjunctival approach for the exposure and osteotomy of traumatic orbitozygomaticomaxillary deformities.

    In the treatment of post-traumatic deformities of the orbitozygomaticomaxillary complex resulting from trauma, the most appropriate exposure must be used. The choice of exposures includes the bicoronal approach and the periorbital incisions. When the whole orbitozygomatic complex is malpositioned, the bicoronal approach is desirable; this can be combined with buccal and eyelid incisions. However, the bicoronal approach is complicated by a longer duration of operation time, post-surgical scars that tend to show, and potential damage to the temporal branch of the facial nerve. A new approach using a C-shape extended transconjunctival approach is possible to have one field of vision to osteotomize the frontozygomatic suture, the lateral orbital wall, inferior orbital rim, lateral maxillary buttress, and zygomatic arch. It takes less operating time and the post-surgical scars are shorter than the bicoronal approach.
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3/15. Neuroparalytic keratitis: a rare manifestation of posttraumatic superior orbital fissure syndrome.

    We present a case of zygomatic fracture complicated with superior orbital fissure syndrome, wherein nasociliary nerve was found involved as a part of the syndrome manifested clinically with neuroparalytic keratitis. The corneal hypoesthesia improved along with other symptoms of superior orbital fissure syndrome after conservative management within 2 months.
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4/15. classification and treatment of zygomatic fractures: a review of 1,025 cases.

    The treatment of zygomatic fractures varies among surgeons, and the cosmetic and functional results are frequently less than optimal. A treatment guideline based on a simple classification of zygomatic fractures is presented. The emphasis is placed on the indications for closed and open reduction, consistent methods of three-dimensional alignment and fixation, and the management of concomitant infraorbital rim and orbital floor fractures. Postoperative results with regard to infraorbital nerve and maxillary sinus dysfunction, malar asymmetry, and orbital complications in the treatment of 1,025 consecutive zygomatic fractures are presented.
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5/15. optic nerve blindness following a malar fracture.

    optic nerve blindness following a malar fracture is an uncommon and usually permanent complication. When the loss of vision is immediate and total, the prognosis is poor. The case of a patient who suffered immediate and complete loss of vision after a malar fracture is presented. Computed tomography revealed compression of the optic nerve by bony fragments. No improvement was observed after megadose steroids and surgical treatment. The incidence, pathogenesis, diagnostic approach and therapeutic possibilities are discussed and the importance of establishing precisely the moment of the loss of vision is stressed.
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6/15. bradycardia and the trigeminal nerve.

    A review of the literature suggests that the oculocardiac reflex is part of a wider phenomenon involving any structure supplied by the trigeminal nerve. This can lead to potentially fatal complications in maxillofacial surgery. A review of the literature is discussed and two further cases are reported.
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7/15. optic nerve decompression via the lateral facial approach.

    Two cases of visual loss after lateral orbital wall fracture are presented: one with retrobulbar hematoma and evidence of optic nerve compression who failed to respond to lateral canthotomy and high-dose corticosteroid administration, and the second with immediate, total blindness associated with fracture of the bony optic canal. In both, extradural decompression of the orbit and optic nerve was achieved through the lateral facial approach with partial return of visual acuity and without surgical complications. The role of orbital and optic nerve decompression in the management of patients with blindness following orbital trauma is controversial. Orbital decompression may be of value for cases of post-traumatic visual loss unresponsive to medical management. If optic nerve injury is suspected as the cause, the additional step of decompression of the optic nerve is a logical but unproven procedure. The indications for optic nerve decompression are not established and should be considered only within the context of the specific needs of the individual patient.
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8/15. Monocular blindness secondary to a non-displaced malar fracture.

    We report a case of monocular blindness subsequent to a non-displaced malar fracture. Injury to the optic nerve, secondary to orbital apex syndrome, was implicated as the cause of blindness. Although most non-displaced malar fractures require no surgical intervention and resolve quite uneventfully, they can be associated with significant morbidity. patients diagnosed as having non-displaced malar fractures should be followed on admission by careful observation and ophthalmologic consultation.
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9/15. Sudden blindness following reduction of a malar fracture.

    A case of reduction of a displaced zygomatic fracture with postoperative proptosis, a dilated, nonreactive pupil, and blindness is presented. early diagnosis with extended lateral canthotomy and removal of the lateral bony wall of the orbit for a depth of 18 mm produced immediate decompression with relief of the proptosis and possible blindness from pressure on the optic nerve. Vision was maintained, and a satisfactory cosmetic result was achieved.
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10/15. Gaze-evoked blindness.

    Three patients with orbital lesions had substantial visual loss while maintaining their eye in an eccentric position of gaze (gaze-evoked blindness). The acuity rapidly returned to baseline levels on return to primary position. Two patients had optic nerve sheath meningiomas and one a displaced portion of orbital bone in contact with the globe. Disc abnormalities and/or choroidal folds were present in all three cases. Gaze-evoked blindness is characterized by the presence of an intraconal mass lesion, but proptosis may be subtle or not present at all. Gaze-evoked blindness should be considered in the differential diagnosis of all patients with atypical amaurosis fugax.
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