Cases reported "Myocardial Stunning"

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1/6. Subarachnoid haemorrhage with transient myocardial injury and normal coronary arteries.

    We present a case of cerebral subarachnoid haemorrhage, with T-wave inversions and myocardial akinesia on echocardiography and ventriculography. Acute coronary angiography showed normal arteries. An aneurysm of the right middle cerebral artery was clamped. Echocardiogram was normalized. We discuss coronary spasm as the possible mechanism of myocardial stunning in subarachnoid haemorrhage.
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ranking = 1
keywords = subarachnoid haemorrhage, subarachnoid, haemorrhage
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2/6. myocardial stunning secondary to aneurysmal subarachnoid hemorrhage.

    BACKGROUND: Although subarachnoid hemorrhage (SAH) is often associated with electrocardiographic abnormalities, profound effects on cardiac performance are rare. CASE DESCRIPTION: A 57-year-old woman who developed loss of consciousness, respiratory distress, severe hypotension, and left ventricular hypokinesis with minimal coronary artery disease is described. Despite normal appearance of the coronary arteries on angiography, left ventricle function was so severely depressed that she required intra-aortic balloon pump support for 24 hours. Mental status changes prompted a head computed tomographic (CT) scan, which showed severe SAH and an intraventricular hemorrhage (IVH). cerebral angiography demonstrated a basilar apex aneurysm. An echocardiogram done on hospital day 6 was normal. A left frontal ventriculostomy catheter was placed. This was later changed to a lumbar subarachnoid (SA) drain. The patient underwent an orbito-zygomatic craniotomy and aneurysm clipping. Although several serious medical problems occurred during her hospitalization, at follow-up, her sole neurological impairment was a minimal and resolving oculomotor paresis. CONCLUSION: This patient's respiratory failure and severe hypotension were initially thought to be due to a chemical pneumonitis or a cardiomyopathy. However, her symptoms ultimately proved to be secondary to a ruptured basilar apex aneurysm. The complex relationship of SAH to myocardial stunning, as illustrated by this patient, is discussed.
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ranking = 0.44836675412613
keywords = subarachnoid
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3/6. Management of patients with stunned myocardium associated with subarachnoid hemorrhage.

    Cardiac complications are well known after aneurysmal subarachnoid hemorrhage. Electrocardiographic changes occur in 50% to 100% of such cases. Arrhythmias, left ventricular dysfunction, and frank myocardial infarction are infrequently observed. myocardial infarction must be differentiated from neurogenic stunned myocardium, which is a reversible condition. From 1996 to 2001, 105 patients with aneurysmal subarachnoid hemorrhage underwent endovascular treatment at the University of michigan. Of these, four patients with no history of cardiac disease experienced cardiac failure related to neurogenic stunned myocardium. All had signs of left ventricular dysfunction, electrocardiographic changes, and elevated cardiac enzymes. Three had pulmonary edema at presentation. All were diagnosed with myocardial infarction. One underwent coronary angiography, which was normal. All were considered poor surgical candidates and underwent endovascular treatment of the aneurysms. Three of four patients developed symptomatic vasospasm, and two required balloon angioplasty. Three patients achieved good outcomes. The eldest died from severe vasospasm that was unresponsive to angioplasty. Reversible cardiac failure associated with subarachnoid hemorrhage may be due the neurogenic stunned myocardium. Frequent symptomatic vasospasm occurs, possibly related to poor cardiac output and the inability to optimize hyperdynamic hypervolemic therapy, particularly with compromised volume status. These patients can be treated with endovascular therapy of the aneurysms and balloon angioplasty as needed. With aggressive management, patients can recover from these reversible cardiac complications.
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ranking = 0.52309454648049
keywords = subarachnoid
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4/6. Postictal neurogenic stunned myocardium.

    Neurogenic left ventricular dysfunction is a recognized complication of subarachnoid hemorrhage, but this condition has not been reported after seizure activity. The authors present two cases of neurogenic stunned myocardium after convulsive seizures, suggesting that ictal activity can lead to sympathetically mediated cardiac injury.
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ranking = 0.074727792354355
keywords = subarachnoid
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5/6. Cardiac effects of acute myelitis.

    Neurogenic stunned myocardium has been described in association with subarachnoid hemorrhage, guillain-barre syndrome, and metastatic brain tumors. We describe a case of neurogenic stunned myocardium associated with acute myelitis. A 27-year-old female presented with acute onset of quadriplegia, sensory deficit, and acute pulmonary edema. magnetic resonance imaging was consistent with acute myelitis. Echocardiogram showed left ventricular ejection fraction of 35% with moderate to severe global hypokinesis. During the course of admission, she had several episodes of sinus bradycardia and high degree atrioventricular block. All cardiac abnormalities resolved completely in eight days of admission.
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ranking = 0.074727792354355
keywords = subarachnoid
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6/6. Neurogenic stunned myocardium and transient severe tricuspid regurgitation in a child following nonaccidental head trauma.

    Neurogenic stunned myocardium is widely reported in the adult literature and is typically associated with subarachnoid hemorrhage. We present a case of neurogenic stunned myocardium and transient severe tricuspid regurgitation in a 7-month-old infant following nonaccidental head trauma.
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ranking = 0.074727792354355
keywords = subarachnoid
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