1/8. Unusual CT and MRI appearance of carbon monoxide poisoning.Unilateral low attenuation areas within the right putamen, globus pallidus and thalamus were observed on CT in a patient after exposure to carbon monoxide. A transient bilateral appearance was found on subsequent CT examinations. Hemorrhagic infarction of the right putamen, and ischemic lesions in both thalami were visualized on MRI 2 weeks later.- - - - - - - - - - ranking = 1keywords = putamen (Clic here for more details about this article) |
2/8. Transient tic disorder following carbon monoxide poisoning.We report a 12-year-old male patient who developed transient motor and vocal tics twelve days after carbon monoxide (CO) poisoning. Cranial magnetic resonance image (MRI) of the patient showed bilateral symmetric hyperintensity in the caudate nucleus and putamen. Tic disorder was successfully treated with haloperidol. Thirty-three months after CO poisoning, the patient was asymptomatic and MRI revealed atrophy in caudate nucleus and putamen. The mechanism of tic disorder in CO intoxication is discussed.- - - - - - - - - - ranking = 1keywords = putamen (Clic here for more details about this article) |
3/8. role of contrast enhancement in cerebral CT of carbon monoxide poisoning.Postcontrast cerebral CT offers advantages as an adjunct to noncontrast CT in the evaluation of patients with carbon monoxide intoxication. It is of particular value when the noncontrast CT is normal with its ability to enhance otherwise isodense lesions. In this regard it can more accurately define the extent of the CNS insult and in some instances offer insight into the patient's eventual clinical outcome. Contrast medium was useful in this instance to more clearly elucidate typical injury to globus pallidus as well as rather unusual involvement of putamen.- - - - - - - - - - ranking = 0.5keywords = putamen (Clic here for more details about this article) |
4/8. Delayed choreoathetosis following acute carbon monoxide poisoning.Ten days after accidental exposure to carbon monoxide, a 17-year-old youth developed transitory choreoathetosis of both arms, face, and neck, with moderate dysarthria. CT revealed symmetric bilateral infarction in the head of the caudate nucleus, the putamen, and the small parts of the anterolateral globus pallidus.- - - - - - - - - - ranking = 0.5keywords = putamen (Clic here for more details about this article) |
5/8. Magnetic resonance features in carbon monoxide poisoning.The authors describe unusual magnetic resonance findings due to carbon monoxide poisoning in a 34-year-old woman. With T2-weighted imaging, increased signal intensity was observed bilaterally in the putamen and the caudate nucleus. Lesions of high signal intensity in the globus pallidus, which have been previously reported, were also observed.- - - - - - - - - - ranking = 0.5keywords = putamen (Clic here for more details about this article) |
6/8. Acute hydrocephalus following carbon monoxide poisoning.Carbon monoxide remains a significant cause of poisoning in children. Cerebral edema is often the cause of significant morbidity and mortality in exposed children. While lesions of the basal ganglia have been well documented, the advent of neuroimaging has allowed antemortem demonstration of infarctions of the globus pallidus and putamen with carbon monoxide intoxication. Acute hydrocephalus following carbon monoxide poisoning has been a rare occurrence. We report a 2 year 6 month-old boy who, to our knowledge, represents the first reported case in which repeat computed tomography documented the evolution of hydrocephalus due to carbon monoxide exposure in a child.- - - - - - - - - - ranking = 0.5keywords = putamen (Clic here for more details about this article) |
7/8. A long-term follow-up study of serial magnetic resonance images in patients with delayed encephalopathy after acute carbon monoxide poisoning.Serial magnetic resonance (MR) images were obtained for 18 months in the follow-up of a patient with delayed encephalopathy after acute carbon monoxide (CO) poisoning. The characteristic findings on T2-weighted MR images (T2WI) were high intensity in the bilateral periventricular white matter and pallidal regions, and low intensity in the striatum. Although the neurological symptoms had improved by about 5 months after exposure, the high signal intensity in the white matter remained visible for 18 months. In addition, low signal intensity in the putamen seen on T2WI, which suggested iron deposition, was seen throughout the 18 months.- - - - - - - - - - ranking = 0.5keywords = putamen (Clic here for more details about this article) |
8/8. magnetic resonance imaging and 11C-N-methylspiperone/positron emission tomography studies in a patient with the interval form of carbon monoxide poisoning.Magnetic resonance (MR) and (11)C-N-methylspiperone ((11)C-NMSP)/positron emission tomography (PET) imagings were repeatedly performed in a 50-year-old man with the interval form of carbon monoxide (CO) poisoning. In MR images obtained when delayed neuropsychiatric symptoms developed (two months after poisoning), the inner segments of the bilateral globus pallidus appeared as high signal intensities in the T1-weighted and low signal intensities in the T2-weighted images, suggesting prior focal hemorrhage in these areas. A PET study with (11)C-NMSP performed at that time showed an increase in dopamine D2 receptor binding in the caudate and putamen. Treatment with bromocriptine was very effective and five months after the poisoning, MR and (11)C-NMSP/PET images showed improvement, concomitantly with the disappearance of the neuropsychiatric symptoms.- - - - - - - - - - ranking = 0.5keywords = putamen (Clic here for more details about this article) |