How similar are PCOS and non-classical congenital adrenal hyperplasia?

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For almost 14 years it's seemed as though I've had PCOS. One doctor did suggest a possible adrenal disorder instead. I just had lab tests done and this doctor doesn't think I have PCOS(though I've been on Yasmin for years, could that make a difference in the tests?). So I looked up adrenal disorders and CAH came up.
Any ideas of where to look or where to go from here(I'll bring this up at my next ob/gyn appt but I was just curious until then...).
First of all these 2 conditions differ greatly in the causative factors and therefore are treated very differently. CAH is the result of an enzyme deficiency and should be easy to rule out with testing, it is most common in Ashkanazi Jews.

PCOS is known to be related to the insulin/glucose regulation in the body. PCOS is also though to be genetic, although not every woman with PCOS has a known geneic marker for it.

The symptoms of non-classical CAH are similar to PCOS and include rapid growth and premature puberty in early childhood, excessive hair growth, irregular menstrual periods, acne, and sometimes, infertility in either males or females.

As part of the standard diagnostic procedure for PCOS most physicians will run an adrenal function test (mine did) in order to rule out the less likely adrenal issues. Birth control pills should not have an impact on Adrenal testing but will have in impact on your hormone levels, lining thinckness and number of cysts in your ovaries.

If you were diagnosed as having the presence of multiple cysts in your ovaries accompanied with other symptoms of PCOS (hair growth, absence of periods etc.), glucose tolerance, insulin intolerance or elevated testosterone levels then most likely your initial diagnosis of PCOS is correct. However if you were never tested for adrenal issues you should be, as the treatment for PCOS and non-classical CAH differ markedly. Current reccomendations are that a diagnosis of PCOS should only be made after ruling out other possibilities.  (+ info)

I was told during a biospy of my breast that I had atypical hyperplasia. Please help me?

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I heard that there is a high risk of getting cancer with atypical hyperplasia. I am taking tamoxifen, but is that the answer? What should I do, or what can a Dr. do to find out if it has turned cancerous?
Atypical hyperplasia in breast is associated with increased risk of developing breast cancer. Some studies showed that tamoxifen reduce that risk. Tamoxifen seems to make sense especially in patients with family history of gene associated with breast cancer. Tamoxifen also gives certain side effects such as hot flash, blood clots, stroke and endometrial cancer.
Your doctor should already tell you about the follow up care. They probably want to see you back for exam and mammogram at least every year or two.
Good luck.  (+ info)

A recent hysteroscopy has shown microglandular hyperplasia. What does this mean.?

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I have been told that no malignant cells have been seen but that I need to see an oncologist urgently. Why do I need to see an oncologist if there are no malignant cells?
hyperplasia isnt normal either dear. it means there r too many cells in ur uterine lining.. its not cancer but stil it cud become tht later on. it depends on ur age n the pathology report.
try to read up on the topic n write down all the q's tht come to u on a piece of paper b4 u c ur doc. he/she wil answer them.
take some1 along with u like ur mom or sis or husband..  (+ info)

whats the pathophysiology of benign prostatic hyperplasia ?

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whats the pathophysiology of benign prostatic hyperplasia? it is more easier for me to understand a diagramed pathophysiology of the said disease.. but narrative form is ok.. thanks for helping..
jazz- Because the answer to your question is very long, I'll try to share some important features. The male prostate gland is located below the bladder. The seminal vesicles (containing sperm in semen) are located posterior to the prostate. The urethra exits from the bladder and goes through the prostate before exiting to the penile urethra (pee hoile).

The normal prostate is composed of glands and stroma. The glands are lined mucin-secreting epithelium. The fibromuscular stroma between the glands accounts for about half of the volume of the prostate. A prostate is about the size of a walnut.

Acute prostatitis (inflammation) is not common. Causative agents include bacteria similar to those causing urinary tract infections, as well as Neisseria gonorrheae. A related complication of prostatic abscess is uncommon. The edema and slight enlargement of the prostate with acute inflammation do not generally cause major symptoms, but may be associated with some pain on urination.

Chronic prostatitis may follow acute prostatitis. In some cases, bacteria can be cultured from urine that indicate the cause. In other cases, chlamydial organisms may be the cause. In some cases, no organism can be identified as a cause. Symptoms of painful urination along with low grade pelvic pain or low back pain may be present.

Nodular prostatic hyperplasia ((enlargement termed benign prostatic hyperplasia, or BPH) is a common condition as men age. Perhaps a fourth of men have some degree of hyperplasia by the fifth decade of life. By the eighth decade, over 90% of males will have prostatic hyperplasia. However, in only a minority of cases (about 10%) will this hyperplasia be symptomatic and severe enough to require surgical or medical therapy.  (+ info)

Can you get pregnant after having endometrial hyperplasia?

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Yes You Can.
it may take alittle longer
usually woman with endometrial hyperplasia has a high level of estrogen and a low level of progestrone
you could use herbs to balance the hormones out or go to the doctors and get a prescription.  (+ info)

Thymic hyperplasia post hodgkins treatment?

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Does anybody know about thymic hyperplasia after hodgkins treatment. Is it a big deal?
The thymus is a gland that at birth is rather large and produces T-cells for your immune system. Over time as your body learns new ways/other ways to fight infection the thymus shrinks and virtually disappears. With the treatment of Hodgkin's thymic hyperplasia can sometimes occur. It is when the thymus enlarges from stress on the body from the treatment and/or the treatment itself. It is usually benign and should go away on it's own, however your doctor should monitor it.

Hope that helps.  (+ info)

Is Prometrium safe to take and is anyone taking it for simple hyperplasia?

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It should be totally fine to take as long as you aren't allergic to peanuts (it contains peanut oil). As you know, with any medication, there are always risks. However, the risk of not managing your simple hyperplasia is far greater - namely, endometrial cancer. Of course, I'm sure your healthcare provider already explained this to you. :-) If you are not already planning on it, I'd suggest requesting periodic transvaginal ultrasounds to make sure the endometrial stripe (lining) is staying nice and thin immediately after you cycle on the prometrium. Best of luck to you!  (+ info)

What is pseudo angiomatosis stromal hyperplasia?

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go to:
http://www.jpgmonline.com/article.asp?issn=0022-3859;year=1997;volume=43;issue=2;spage=50;epage=1;aulast=Deshmukh  (+ info)

Hello! Does anyone have something called focal nodular hyperplasia? Its a benign growth on the liver?

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I'm just curious to see if anyone else has had it and details about it etc. Like did you have surgery to get it removed or did their diet impact on what happened? Did you get pains at all? Its usually found by accident by an ultrasound or CT scan thats being done for other problems like stomach pains or problem with gall bladder etc. Mine was found on a ultrasound and i'm waiting to see a specialist. Any help would be really great! I'm just scared about what it might be or if I have to get surgery. Mine is pretty big (7.5 x 6.5 cm) and i've been getting really bad pains after i eat etc. So i think i may have to get it removed.

Thanks! ;-)
It is a very common thing and you dont have to worry...

Read on....

Focal nodular hyperplasia (FNH) is the second most common tumor of the liver, surpassed in prevalence only by hepatic hemangioma. FNH is believed to occur as a result of a localized hepatocyte response to an underlying congenital arteriovenous malformation. FNH is a hyperplastic process in which all the normal constituents of the liver are present but in an abnormally organized pattern. Results of liver function tests in these patients usually are within the reference range.

While the use of contraceptive agents is not implicated in the pathogenesis of FNH, their use is associated with an increased rate of complications in patients with FNH, and they may be a factor in the development of FNH. In symptomatic females, hemorrhagic foci or infarctions may occur within the FNH; these are aggravated by administration of contraceptive agents. The rare complication of a spontaneous rupture into the peritoneum has also been associated with contraceptive use.

...... To know more ... Visit.  (+ info)

What is Lymphoid Hyperplasia?

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I have a lot of stomach problems. I recently got an upper GI and small bowel series. I definately have IBS. Now the gastorenterologist saw something in the x-ray that he called lymphoid hyperplasia. I am not sure what it means and he also said the term is rare. He said that this was found in the distal illeum. I got a colonoscopy which was normal. He asked for me to get a capsule endoscopy because you can only view the small bowel closely this way. I havent found a doctor to give me that test. Should I be worried, and what is it?
Here this might help you out:
Lymphoid hyperplasia of the intestines is a benign reactive process also known as pseudolymphoma, lymphonodular hyperplasia, or terminal lymphoid ileitis.1 It occurs in all age groups, but is best described in children.1,2 There are no definitions or valid criteria for when normal lymphoid tissue becomes hyperplastic, or for when hyperplasia becomes pathologic.3 The dividing line between physiologic nodules and hyperplasia is thus ill-defined. Lymphoid hyperplasia may be discovered incidentally, at colonoscopy, or in patients undergoing colectomy, especially in the last few centimeters of the terminal ileum. However, cases that are symptomatic due to intussusception or prolapse are well documented.12 Lymphoid hyperplasia occurs mainly in the rectum and in the ileocecal region.1 Eighty percent of lymphoid polyps are sessile and solitary; the remainder are pedunculated and/or multiple (numbering 2 to 6).

Several causes of lymphoid hyperplasia have been postulated. Giardin lamblia is often present. In a small minority, immunodeficiencies underlie the hyperplastic state.2 Some patients have low or absent IgA and IgM levels, decreased IgG levels, susceptibility to infection, and diarrhea with or without steatorrhea. There is also an association with familial adenomatous polyposis and Gardner syndrome. In children, lymphoid hyperplasia is often associated with viral infection.

The pathology of lymphoid hyperplasia is quite characteristic, consisting of prominent lymphoid follicles with active germinal centers located in the mucosa and submucosa. The most important differential diagnosis is with lymphomatous polyposis. The presence of highly reactive germinal centers, numerous cell types, prominent vascularity, and polyclonality as determined immunohistochemically are the most important features in the differential diagnosis with lymphoma. Lymphoid hyperplasia may be differentiated from follicular lymphoma presenting as lymphomatous polyposis by Bcl-2 immunostaining of follicular germinal centers.

The clinical significance of lymphoid hyperplasia lies in the possibility of these nodules serving as a nidus for prolapse and intussusception and in the association with immunosuppressive states. Local excision is curative; occasionally, spontaneous remission has been noted.  (+ info)

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