What are some exercises to help uteine prolapse?

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I have heard that a change in diet and certain exercises can help to improve uterine prolapse. I believe I have a mild case (just starting to see a bulge). I won't see my doctor for two months as that is the earliest appointment I can get. I am interested in reading up on the diet and exercises if someone could suggest a site or something.
Pelvic floor exercises

See the wiki and Anna Hayward's excellent site!  (+ info)

What is the best alternative way to treat Mitral Valve Prolapse?

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I was diagnosed with Mitral valve prolapse yrs. ago, but this has got worse and very troublesome and is interfering with daily routines stay inside away from people because of panic attacks and anxiety feet legs ,hands, and under eyes swell up like b***ons and causes discomfort.I sweat *** the time and catch infections very easly.Is their any over the counter medicenes or vitamins that will help my condition?
Mitral valve prolapse is a structural abnormality of the heart. Over the counter self-treatment is not going to be effective for what can be a very serious problem.

The panic attacks that you describe are not a part of MVP. That sounds like a separate anxiety-related issue.

You need to see a physician for a thorough history and physical, plus cardiac workup to evaluate the severity of your problem, and to distinguish the cardiac issues from the anxiety issues. You may need to be on prescription medications to optimize your heart function. The last resort for mitral valve disease is valve replacement surgery.

People with MVP are susceptible to SBE - subacute bacterial endocarditis, which is an infection of the heart. It is difficult to treat and can lead to further heart problems.

If you let things go too long, your condition may progress to the point where little can be done. Make an appointment with a doctor ASAP and get yourself sorted out. This is nothing to fool around with.  (+ info)

What is the best cardio exercise for eliminating visceral fat? Anaerobic or Aerobic cardio?

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That dangerous bellyfat that is around the heart and is more of a hard feeling of fat around the abdomen. I have to get rid of it because of all the dangers that it presents. Which type of cardio should I be doing and what types of foods should I be eating to gradually lower visceral fat out of my abdomen.
Aerobic definitely.

Also eat a low carb diet. Avoid potatos, saturated fats and try to eat food rich in omega 3, fruit, vegs, nuts and pulses.

Drink green tea two or three times a day, it's really good for your heart and circulation.  (+ info)

How can I tell if my stomach is made up of visceral fat, or subcutaneous fat?

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I've been reading about visceral fat, the deadly fat that wraps around your organs, and how dangerous it is. I want to know how I can tell if my big stomach is made up mostly of visceral or subcutaneous fat.

I do have a big stomach, I'm 215 pounds and I'm 5'4, but it seems to be mostly soft fat, not the hard round belly you see on old men. Does this mean my fat is mostly subcutaneous?
Its still fat so you want to get rid of it.  (+ info)

Which insurance company claims disk prolapse operation?

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I am planning to take a health insurance policy for my parents. I also want to know which insurance company will claim the disk prolapse operation? Because my dad is suffering from disk back pain. I'd like to take such a plan. I am from pune. Working in software industry. My company is not providing for total family, but providing for me.
If you do buy a private plan, you father would most likely have to wait out a pre-existing clause exemption. This means he may have to wait a year or two before any surgery, or any medical care related to his back for that matter, would be covered. However, since you are not protected by the statutes of a group policy (those policies offered by an employer are often much more liberal), there is a possibility that anything ever related to his back would NOT be covered. This prevents people from buying insurance just to get covered for an expensive procedure and then ditching the coverage afterwards.

Aside from that, just about any major medical plan will cover back surgery, but for how much and under what limitations are going to depend on the personal provisions you elect to pay for in the policy. For someone with a known medical condition, I'd expect to pay well over $1,000 a month (probably close to 2) for his coverage on a good, private policy, and then have to wait a year or two to wait out any pre-existing clauses.  (+ info)

Is it safe to get a tattoo if you have Mitral Valve Prolapse?

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My mom wants to get her first tattoo, but she has a heart condition called Mitral Valve Prolapse. It's sort of like a really bad heart murmer. At one time, a doctor told her that she should have penicillin before having anything done (like dental work). Would that apply to having a tattoo?
Absolutely, she needs to get antibiotics from her family Dr. because she is at a very high risk to develop endocarditus, which is an infection involving her heart. She must speak with her family Dr. first.!!  (+ info)

Is it safe for someone with a mitral valve prolapse to have children?

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I have mitral valve prolapse (which means that my mitral valve doesn't close properly). I am not ready to have children yet. I was just wanting some information before I have kids.
The mitral valve is located in the heart. It means you have a broken heart. You can't love, and you need to find love to have children. Sorry, sister, you're out of luck.

Just playing with you. Take care of your heart, and your doctor will tell you if you can handle the strain on the heart from pregnancy. It may be unlikely, but you can always adopt.  (+ info)

My doctor prescribed me physical therapy for mild bladder prolapse. What will they do there?

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And will it make a difference? Will it cure the prolapse or just make my pelvic floor stronger? Is this a lifelong condition?
The physical therapist should definetely be someone who specializes in this treatment...not every clinic offers this, so you may need to do your homework.

The initial evaluation will focus on a history of your problem and well as an examination. It might include an internal examination and possible use of force tranducers to establish the strength of her pelvic floor. The aim of therapy is usually to reduce or eliminate episodes of incontinence or pelvic pain.

As to your actual prognosis, this would best be assessed by the PT evaluating you.  (+ info)

how much does it cost to repair a Mitral Valve Prolapse?

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Mitral Valve Prolapse (MVP) is a heart defect that can be repaired either by laser surgery (non-open surgery) or by open surgery. How much does this cost with or without insurance. I hope to get insurance soon. Is a heart murmur repairable?
Mitral Valve Repair Surgeons & Hospitals in India
India has gained a name for high tech medical surgeries & it is now quite comment for many in the west to consider mitral valve prolapse repair in India. Many of the surgeon have returned to India from training in leading centers in the USA 7 UK.

India's leading cardiologists and cardo-thoracic surgeons combined with adoption of the latest technology have given it the edge over other competing medical tourism destinations in case of heart surgeries. Of course, needless to say, it is also the most affordable.

With skyrocketing costs of medical care in the US, going abroad for heart surgery is becoming very common. The cost of mitral valve repair in the US can be anywhere between $40,000 - $200,000. The same procedure in India can be done for about $10,000.

http://www.medicaltourismco.com/cardiology/mitral-valve-repair-replacement.php  (+ info)

What is remedy for Mitral valve prolapse?

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Report of 2D Echo of my son aged 20 shows Mitral valve prolapse. No rheumatic afflication. LVEF 60%. No pulmonory hypertension. No effusion/clot. Test was done on 14-10-06. Actually he has pain in knees and lower back since 8 year. After several tests it was diagnosed as Ankylosing Spondylitis two years back. Please help us in diagnosis and suggest the treatment.
Ankylosing spondylitis is a form of chronic inflammation of the spine and the sacroiliac joints. The sacroiliac joints are located in the low back where the sacrum (the bone directly above the tailbone) meets the iliac bones (bones on either side of the upper buttocks). Chronic inflammation in these areas causes pain and stiffness in and around the spine. Over time, chronic spinal inflammation (spondylitis) can lead to a complete cementing together (fusion) of the vertebrae, a process referred to as ankylosis. Ankylosis leads to loss of mobility of the spine.

Ankylosing spondylitis is also a systemic rheumatic disease, meaning it can affect other tissues throughout the body. Accordingly, it can cause inflammation in or injury to other joints away from the spine, as well as other organs, such as the eyes, heart, lungs, and kidneys. Ankylosing spondylitis shares many features with several other arthritis conditions, such as psoriatic arthritis, reactive arthritis, and arthritis associated with Crohn's disease and ulcerative colitis. Each of these arthritic conditions can cause disease and inflammation in the spine, other joints, eyes, skin, mouth, and various organs. In view of their similarities and tendency to cause inflammation of the spine, these conditions are collectively referred to as "spondyloarthropathies." For more information, please read the following articles; Psoriatic Arthritis, Reactive Arthritis, Crohn's Disease and Ulcerative Colitis.

Ankylosing spondylitis is 2-3 times more common in males than in females. In women, joints away from the spine are more frequently affected than in men. Ankylosing spondylitis affects all age groups, including children. The most common age of onset of symptoms is in the second and third decades of life.


What causes ankylosing spondylitis?

The tendency to develop ankylosing spondylitis is believed to be genetically inherited, and the majority (nearly 90%) of patients with ankylosing spondylitis are born with the HLA-B27 gene. Blood tests have been developed to detect the HLA-B27 gene marker, and have furthered our understanding of the relationship between HLA-B27 and ankylosing spondylitis. The HLA-B27 gene appears only to increase the tendency of developing ankylosing spondylitis, while some additional factor(s), perhaps environmental, are necessary for the disease to appear or become expressed. For example, while 7% of the United States population have the HLA-B27 gene, only 1% of the population actually have the disease ankylosing spondylitis. In Northern Scandinavia (Lapland), 1.8% of the population have ankylosing spondylitis while 24% of the general population have the HLA-B27 gene. Even among HLA-B27 positive individuals, the risk of developing ankylosing spondylitis appears to be further related to heredity. In HLA-B27 positive individuals who have relatives with the disease, their risk of developing ankylosing spondylitis is 12% (6 times greater than for those whose relatives do not have ankylosing spondylitis).
Ankylosing spondylitis is a form of chronic inflammation of the spine and the sacroiliac joints. The sacroiliac joints are located in the low back where the sacrum (the bone directly above the tailbone) meets the iliac bones (bones on either side of the upper buttocks). Chronic inflammation in these areas causes pain and stiffness in and around the spine. Over time, chronic spinal inflammation (spondylitis) can lead to a complete cementing together (fusion) of the vertebrae, a process referred to as ankylosis. Ankylosis leads to loss of mobility of the spine.

Ankylosing spondylitis is also a systemic rheumatic disease, meaning it can affect other tissues throughout the body. Accordingly, it can cause inflammation in or injury to other joints away from the spine, as well as other organs, such as the eyes, heart, lungs, and kidneys. Ankylosing spondylitis shares many features with several other arthritis conditions, such as psoriatic arthritis, reactive arthritis, and arthritis associated with Crohn's disease and ulcerative colitis. Each of these arthritic conditions can cause disease and inflammation in the spine, other joints, eyes, skin, mouth, and various organs. In view of their similarities and tendency to cause inflammation of the spine, these conditions are collectively referred to as "spondyloarthropathies." For more information, please read the following articles; Psoriatic Arthritis, Reactive Arthritis, Crohn's Disease and Ulcerative Colitis.

Ankylosing spondylitis is 2-3 times more common in males than in females. In women, joints away from the spine are more frequently affected than in men. Ankylosing spondylitis affects all age groups, including children. The most common age of onset of symptoms is in the second and third decades of life.


What causes ankylosing spondylitis?

The tendency to develop ankylosing spondylitis is believed to be genetically inherited, and the majority (nearly 90%) of patients with ankylosing spondylitis are born with the HLA-B27 gene. Blood tests have been developed to detect the HLA-B27 gene marker, and have furthered our understanding of the relationship between HLA-B27 and ankylosing spondylitis. The HLA-B27 gene appears only to increase the tendency of developing ankylosing spondylitis, while some additional factor(s), perhaps environmental, are necessary for the disease to appear or become expressed. For example, while 7% of the United States population have the HLA-B27 gene, only 1% of the population actually have the disease ankylosing spondylitis. In Northern Scandinavia (Lapland), 1.8% of the population have ankylosing spondylitis while 24% of the general population have the HLA-B27 gene. Even among HLA-B27 positive individuals, the risk of developing ankylosing spondylitis appears to be further related to heredity. In HLA-B27 positive individuals who have relatives with the disease, their risk of developing ankylosing spondylitis is 12% (6 times greater than for those whose relatives do not have ankylosing spondylitis).

Ankylosing spondylitis is a form of chronic inflammation of the spine and the sacroiliac joints. The sacroiliac joints are located in the low back where the sacrum (the bone directly above the tailbone) meets the iliac bones (bones on either side of the upper buttocks). Chronic inflammation in these areas causes pain and stiffness in and around the spine. Over time, chronic spinal inflammation (spondylitis) can lead to a complete cementing together (fusion) of the vertebrae, a process referred to as ankylosis. Ankylosis leads to loss of mobility of the spine.

Ankylosing spondylitis is also a systemic rheumatic disease, meaning it can affect other tissues throughout the body. Accordingly, it can cause inflammation in or injury to other joints away from the spine, as well as other organs, such as the eyes, heart, lungs, and kidneys. Ankylosing spondylitis shares many features with several other arthritis conditions, such as psoriatic arthritis, reactive arthritis, and arthritis associated with Crohn's disease and ulcerative colitis. Each of these arthritic conditions can cause disease and inflammation in the spine, other joints, eyes, skin, mouth, and various organs. In view of their similarities and tendency to cause inflammation of the spine, these conditions are collectively referred to as "spondyloarthropathies." For more information, please read the following articles; Psoriatic Arthritis, Reactive Arthritis, Crohn's Disease and Ulcerative Colitis.

Ankylosing spondylitis is 2-3 times more common in males than in females. In women, joints away from the spine are more frequently affected than in men. Ankylosing spondylitis affects all age groups, including children. The most common age of onset of symptoms is in the second and third decades of life.


What causes ankylosing spondylitis?

The tendency to develop ankylosing spondylitis is believed to be genetically inherited, and the majority (nearly 90%) of patients with ankylosing spondylitis are born with the HLA-B27 gene. Blood tests have been developed to detect the HLA-B27 gene marker, and have furthered our understanding of the relationship between HLA-B27 and ankylosing spondylitis. The HLA-B27 gene appears only to increase the tendency of developing ankylosing spondylitis, while some additional factor(s), perhaps environmental, are necessary for the disease to appear or become expressed. For example, while 7% of the United States population have the HLA-B27 gene, only 1% of the population actually have the disease ankylosing spondylitis. In Northern Scandinavia (Lapland), 1.8% of the population have ankylosing spondylitis while 24% of the general population have the HLA-B27 gene. Even among HLA-B27 positive individuals, the risk of developing ankylosing spondylitis appears to be further related to heredity. In HLA-B27 positive individuals who have relatives with the disease, their risk of developing ankylosing spondylitis is 12% (6 times greater than for those whose relatives do not have ankylosing spondylitis).


The mitral valve (also known as the bicuspid valve or left atrioventricular valve), is a dual flap (bi = 2) valve in the heart that lies between the left atrium (LA) and the left ventricle (LV). In Latin, the term mitral means shaped like a miter, or bishop's cap. The mitral valve and the tricuspid valve are known collectively as the atrioventricular valves because they lie between the atria and the ventricles of the heart and control flow.

A normally functioning mitral valve opens to pressure from the superior surface of the valve, allowing blood to flow into the left ventricle during left atria systole (contraction), and closes at the end of atrial contraction to prevent blood from back flowing into the atria during left ventricle systole. In a normal cardiac cycle, the atria contracts first, filling the ventricle. At the end of ventricular diastole, the bicuspid valve shuts, and prevents backflow as the ventricle begins its systolic phase. Backflow may occur if the patient suffers from mitral valve prolapse, causing an audible "murmur" during auscultation.


[edit] Anatomy
The mitral valve has two cusps/leaflets (the anteromedial leaflet and the posterolateral leaflet) which guards the opening. The opening is surrounded by a fibrous ring known as the mitral valve annulus. (The orientation of the two leaflets were once thought to resemble a bishop's miter, which is where the valve receives its name.[1]) The anterior cusp protects approximately two-thirds of the valve (imagine a crescent moon within the circle, where the crescent represents the posterior cusp). These valve leaflets are prevented from prolapsing into the left atrium by the action of tendons attached to the posterior surface of the valve, chordae tendinae.

The inelastic chordae tendineae are attached at one end to the papillary muscles and the other to the valve cusps. Papillary muscles are finger like projections from the wall of the left ventricle. Chordae tendinae from each muscle are attached to both leaflets of the mitral valve. Thus when the ventricle contracts, the intraventricular pressure forces the valve to close, while the tendons prevent the valve from opening in the wrong direction.


[edit] Normal physiology
During left ventricular diastole, after the pressure drops in the left ventricle due to relaxation of the ventricular myocardium, the mitral valve opens, and blood travels from the left atrium to the left ventricle. About 70-80% of the blood that travels across the mitral valve occurs during the early filling phase of the left ventricle. This early filling phase is due to active relaxation of the ventricular myocardium, causing a pressure gradient that allows a rapid flow of blood from the left atrium, across the mitral valve. This early filling across the mitral valve is seen on doppler echocardiography of the mitral valve as the E wave.

After the E wave, there is a period of slow filling of the ventricle.

Left atrial contraction (left atrial systole) (during left ventricular diastole) causes added blood to flow across the mitral valve immediately before left ventricular systole. This late flow across the open mitral valve is seen on doppler echocardiography of the mitral valve as the A wave. The late filling of the LV contributes about 20% to the volume in the left ventricle prior to ventricular systole, and is known as the atrial kick.  (+ info)

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