I am suffering in juvenile myoclonic epilepsy for 17 years.?

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Now i in keppra ( levetiracetum-500), and take daily 1500 for last two months. But i fell very weak, depressed, angry, cant keep my concentration.After starting the medicine, two times i become fent after convultion. Is there anyone who can help me?
Unfortunately finding the right medication and dosage for control of your epilepsy is done by trial and error. You need to tell your doctor what your experiencing and he may up or down your dosage or may find an alternative.
I just got taken off Depakote (500mg 2 a day) and now almost 5 months seizure free. Heres hoping that continues.
I went thru alot of meds before they came to Depakote! Phenobarbitol, (SP) Lorazapam, Felbatol and mixtures of them.  (+ info)

impression: global symmetric decreased uptake in the cerebellar hemispheres,medial portions of temporal lobes?

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and mild decreased uptake in the anterior portions of the parietal lobes. I have vit. B12 def. what does it mean?
subacute combined degeneration neuropathy from b-12 deficiency  (+ info)

could i die from juvenile myoclonic epilepsy?

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could i die from juvenile myoclonic epilepsy?
No. It won't kill you.

For a little reassurance and some more information, see the following Web site: http://www.epilepsy.com/epilepsy/epilepsy_juvenilemyoclonic.htm.l  (+ info)

My brain gets these little shivers - Myoclonic Jerks?

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For the last few months, I've been getting this odd sensation in my brain that feels just like the little myoclonic jerks you get in your muscles. I'm not really sure what this is from. I am not on and have never been on anti-depressants or any other drugs. I usually get these zaps while I'm driving, and when I'm sewing in low light. It's like my brain is twitching. What's going on?
Try upping your vitamin B12 levels and see how you go. B12 deficiency affects the nerves. I would also up your calcium levels too as it affects the muscles..well your brain isn't a muscle lol but hey try it anyway lol. :)  (+ info)

Can Myoclonic Epilepsies kill people ? how much does it exactly hurts people ?

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I wanna knw more.Please dont google it and paste the whole definition here.thx.

Is it one of d worst disease ever or can it be taken ?

Please tell me d side effects of this desease..

Thx alot...
HOW TO CURE THIS DESEASE ?sorry forgot to ask..
Myclonic epilepsy shouldn't hurt anyone unless they fall or hit something as a result of a seizure. Epilepsy doesn't really have a "cure," but with medication it can be controlled.  (+ info)

Does anyone know a cure for cerebellar ataxia?

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A friend is experiencing this baffling condition and the docs don't know what is causing it. They believe his cerebellum is shrinking causing coordination problems, speech impairment/slurring, unsteady gait. He does not know anyone with the condition. It seems to be progressing and he's been told by docs he could end up in a wheel chair. As a child he had Gilliam Beret disease, a disease that destroys nerve tissue. He was paralyzed at age 13, had therapy and fully recovered. He is now 45 years of age, otherwise healthy--perhaps intolerant of wheat and dairy, but a healthy European male living in the U.S.
There is no known cure for cerebellar ataxia.  (+ info)

Is it possible to outgrow Juvenile Myoclonic Epilepsy?

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Please help. I'm only 18 years old..
Unfortunately juvenile myclonic epilepsy is one that is not likely to be outgrown. In about 80% of cases though it is controlled with medication, so be sure to take it on time if you have one prescribed. The only other things you can do are try to avoid triggers like sleep deprivation, alcohol, and stress.  (+ info)

Does anyone know of or had myoclonic seizures growing up? ?

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My son is 14 months old and diagnosed with myoclonic seizures at 11 months old. I wanted to know if you had them or know someone that has had them and the outcome? How old were you when they started? He is about to start Depakote next week. We are weaning him from Zinisomide (Zenogran) now. Any help would be appreciated, thanks.
  (+ info)

Any difference between myoclonic jerks and myoclonic siezures?

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Myoclonus(medical term) or myoclonica jerks or myoclonic siezuers are all one and the same. It is brief, involuntary twitching of a muscle or a group of muscles. It describes a symptom and, generally, is not a diagnosis of a disease. The myoclonic twitches or jerks are usually caused by sudden muscle contractions; they also can result from brief lapses of contraction. Contractions are called positive myoclonus; relaxations are called negative myoclonus. The most common time for people to encounter them is while falling asleep ("sleep starts"), but myoclonic jerks are also a symptom of a number of neurological disorders. Hiccups are also a kind of myoclonic jerk specifically affecting the diaphragm.

Myoclonic jerks may occur alone or in sequence, in a pattern or without pattern. They may occur infrequently or many times each minute. Most often, myoclonus is one of several symptoms in a wide variety of nervous system disorders such as multiple sclerosis, Parkinson's disease, Alzheimer's disease, and Creutzfeldt-Jakob disease.

Anatomically, myoclonus may originate from lesions of the cortex, subcortex or spinal cord. The presence of myoclonus above the foramen magnum effectively excludes spinal myoclonus, but further localisation relies on further investigation with electromyography (EMG) and electroencephalography (EEG).  (+ info)

What is Juvenile Myoclonic Epilepsy?

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A friend of mine has a son that has recently been diagnosed with JME and I've been reading up on it, but I am still slightly confused. From what I understand they can have three different types of seizures each type different. Am I correct?
Juvenile myoclonic epilepsy: diagnosis and treatment
myoclonic epilepsy occurs in up to 10 percent of patients with epilepsy. Though characterized by the triad of absence seizures, myoclonic jerks and generalized tonic-clonic seizures, it is frequently misdiagnosed. Grunewald and Panayiotopoulos review the diagnosis and treatment of juvenile myoclonic epilepsy.

The onset of juvenile myoclonic epilepsy is age-related, with absence siezures beginning between five and 16 years of age. Seizures that begin after 10 years of age are generally mild and may not cause abnormal physical activity. Myoclonic jerks begin an average of four years later and are usually bilateral. Consciousness is not impaired during this activity. Myoclonic jerks may rarely be the only clinical manifestation of juvenile myoclonic epilepsy. Generalized tonic-clonic seizure activity usually begins within a few months to two years. These seizures occur most often on waking and usually follow a series of myoclonic jerks, which become increasingly severe.

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Myoclonic seizure therapy
A Practical Approach to Uncomplicated Seizures in Children
Genetics and Epilepsy: What We Know Today The myoclonic jerks and generalized tonic-clonic seizures of juvenile myoclonic epilepsy are precipitated by a variety of factors (see table). Absence seizures are less likely to be triggered by these factors.

An electroencephalographic (EEG) tracing in patients with juvenile myoclonic epilepsy shows a generalized spike or multiple spike/slow-wave pattern. These spikes may have a typical "compressed Ws" or wormlike appearance. Most patients also have additional focal EEG abnormalities, which may contribute to diagnostic difficulty. A genetic link in juvenile myoclonic epilepsy is apparent, but the mode of inheritance is unclear.

Several characteristics should raise the physician's index of suspicion for juvenile myoclonic epilepsy, including generalized tonic-clonic seizures that occur in the early morning, seizures that are pooly controlled in patients taking carbamazepine and infrequent seizures associated with alcohol use or sleep deprivation.

Up to 90 percent of patients become seizure-free as a result of accurate diagnosis and appropriate treatment. Valproate is the most effective treatment for juvenile myoclonic epilepsy. Clonazepam (0.5 to 2 mg four times per day) may be added for patients with particularly resistant seizures. Patients should be counseled that the "warning" myoclonic jerks that often herald tonic-clonic seizures may not occur during clonazepam therapy. Second-line anticonvulsants include phenobarbital, primidone and possibly phenytoin. Lifelong treatment is generally necessary and withdrawal of medication may precipitate status epilepticus.

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