How can an aneurysm lead to a blood clot which would cause an infarction???

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A patient suffers from severe leg pain while golfing. And pees discolored urine. He was misdiagnosed with muscle trauma when in reality he had a blocked artery in his leg caused by a popliteal artery aneurysm. By the time the blockage was diagnosed, a great deal of muscle tissue had already died and the patient eventually gets an infarction. My question is, how does a Popliteal Artery Aneurysm lead to a blood clot???
Haha, I know exactly where you're getting this from!! House, right?? (sorry, I just LOVE that show)

Anyways, blood clotting is a big risk with aneurysms. Clots can break off and travel through the bloodstream until they get stuck. Normally, treatment is given quickly, and amputation or invasive measures like that are not required. But if it is not diagnosed soon enough, muscle tissue dies, and the limb may have to be amputated.

If you want a more in-depth explanation, here's a good article: http://www.evtoday.com/AAA/2003%20Files/Popliteal%20Artery%20Aneurysms.html  (+ info)

What is the risk of a less than 2mm cerebral aneurysm?

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I had an MRA and they found a tiny saccular aneurysm measuring less than 2mm maximum diameter directed posteriorly at the origin of left A1 segment of anterior cerebral artery. I am 29 years old. Any general information about aneurysms and the danger of the location would be greatly appreciated.
http://www.healthatoz.com/healthatoz/Atoz/common/standard/transform.jsp?requestURI=/healthatoz/Atoz/ency/cerebral_aneurysm.jsp

Looks like there is a 1% chance each year that it could rupture. Maybe that's similar to the sum of other risks of great bodily injury like driving, going into a convenience store or going to a pizzaria in NY.

But you should find out what kinds of activities might be more dangerous and make sure your blood pressure stays good! Since,you've been to a doctor, you have probaly heard this by now.

Good luck.  (+ info)

Do you know what is wrong my right index and middle finder tips have been numb for the last hour?

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Here is some quick background information: I am 31. I have had 5 shoulder surgeries; 4 to help stabilize my shoulder and the other was to remove my first rib to make room for my artery. I have the beginning of arthritis in my shoulder. Thanks!
it may be carpal tunnel syndrome  (+ info)

the right middle cerebral is blocked and right internal capsule is completely injured.what are symptoms?

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If the right middle cerebral artery is blocked then one would get left sided paralysis, visual neglect (a lack of awareness of objects on the left side of the visual field), apraxia (difficulty performing certain motor tasks), and edema (swelling) in the hands or feet.  (+ info)

What will a partial obstruction in a coronary artery likely cause?

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a. pulmonary embolus
b. hypertension
c. angina attack
d. myocardial infarction
c.

Can we do all of your homework for you?  (+ info)

How will I know if the artery begins to close again?

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I've had a myocardial infarction on whom PTCA+STENT was done 2 1/2 years back.
To understand your question you must understand what a myocardial infarction is.

This term refers to the death of a certain segment of the heart muscle (myocardium), usually the result of a focal complete blockage in one of the main coronary arteries or a branch thereof.

The main cause of myocardial infarction is atherosclerosis in the coronary arteries. This event results in impaired contractility of the heart muscle within seconds, and is initially restricted to the affected segment.

The myocardial ischemia or infarction begins in the endocardium (the inner lining of the heart) and spreads to the epicardium (the outer lining of the heart). Irreversible heart damage will occur if the blockage is complete for at least 15-20 minutes. Irreversible damage occurs maximally in the area at risk, and when the occlusion is maintained for 4-6 hours. Most of the damage occurs in the first 2-3 hours. Restoration of flow within the first 4-5 hours is associated with salvage of the heart muscle, but the salvage is greater if flow is restored in the first 1-2 hours. A major determinant of death and illness is the size of the infarct. Increasing the oxygen supply to the involved site of blockage by coronary reperfusio is more effective in salvaging the myocardium than decreasing oxygen demand.

The onset of acute Q-wave myocardial infarction occurs commonly in the morning hours shortly after arising, when there is increasing adrenergic activity, as well as increased blood fibrinogen levels and increased platelet (blood cell) adhesiveness. Non Q wave infarction does not show this circadian rhythm.

The traditional concept that myocardial infarctions can be classified as transmural or nontransmural on the basis of the presence or absence of Q waves is misleading, since autopsy studies have demonstrated convincingly that pathologic Q waves may be associated with nontransmural infarction and may be absent with transmural infarction. These misnomers have been replaced by the terms Q-wave infarction and nonQ-wave infarction for transmural and nontransmural infarction, respectively.

The evolution of a non-Q-wave infarction is charcterized by a lack of development of an abnormal Q wave and by the appearance of reversible ST-T-wave changes with ST depression that usually returns to normal over a few days, but occasionally is permanent. Differentiation between these two types of infarctions has become entrenched, since there are major differences in their pathogenesis, clinical manifestations, treatment, and prognosis. The initiating events in the pathogenesis of Q-wave and non-Q-wave infarction are thought to be identical, namely, coronary occlusion induced by thrombus superimposed on a plaque together with vasoconstriction.

There is considerable evidence, however, to indicate that in non-Q-wave infarction, early spontaneus reperfusion occurs, the mechanism of which remains uncertain. In contrast, in Q-wave infarction, the coronary occlusion is sustained at least for a long enough period to result in extensive necrosis.
One explanation for early spontaneous reperfusion is the lack of sustained vasoconstriction, which may contribute to ocusion. The evidence supporting the existence of early spontaneous reperfusion in non-Q-wave infarction is as follows:

1. Coronary angiographic studies performed in the early hours after onset show that only 20-30% of patients have complete coronary occlusion of the infarct-related vessels;but for Q-wave infarction it is about 80 to 90%.


2. Infarct size is routinely much less than observed with Q wave infarction, which is consistent with salvage by early reperfusion.

3. Peak plasma CK levels are reached on an average of 12 to 13 h after onset of symptoms, indicating early washout of the enzyme, as opposed to about 27 h after Q-wave infarction.

4. Reperfusion-induced contraction necrosis is extremely common, as it is in patients who undergo early reperfusion induced by thrombolytic therapy.

5. Acute mortality rates are around 2 to 3 percent, compared with 10 percent for Q-wave infarction.

6. The complications are minimal compared with those after a Q-wave infarction.

7. Finally, the long-term prognosis is characterized by recurrent episodes of reinfarction, so that after about 2 years, survival is the same as that after Q-wave infarction.

Quite often with the initial heart attack over half of the patients have significant obstructive atherosclerosis in only one vessel. However, in a recent study two fifths of the patients with acute myocardial infarction had angiographic evidence of multiple complex coronary plaques, which were associated with a less favorable in-hospital course. The presence of these plaques with complex morphologic features is the angiographic hallmark of unstable coronary syndromes and correlates with pathologic plaque and thrombus

I'm not a Doctor... I'm just a Corpsman...  (+ info)

Is the middle vein that runs down your arm?

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A major artery that if cut would lead to death?
If you cut through it, you would die, yes.
Without quick treatment..I guess you could bleed to death.  (+ info)

Do you know the data sheet for examination of a patient of carotid artery stenosis?

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This is a part of vascular surgery.
Carotid artery stenosis may lead to cerebral stroke.
Carotid artery stenosis is when plaque forms in the carotid arteries, constricting the flow of blood to the brain. The carotid arteries are the main arteries coming from your heart to your head. The risk is that, if there is plaque, a small piece could break off and get lodged in a smaller vessel in your brain, causing lack of blood to that part of the brain. This can result in in a TIA, or mini stroke, or can even result in something as serious as a brain bleed. An ultrasound is the simplest and safest method for determining plaque, as well as velocity of the blood flow. Velocity at any point of stenosis is a key factor. I could go on about what range velocities should be in, but that opens up a whole worksheet of math problems that I can't begin to explain here.

It would be best to consult your physician if I did not answer your question here, especially as I am not sure what you mean by data sheet. If you are wanting the math equations for velocity, you can e-mail me and I will send you something.  (+ info)

Would A Middle Aged Man w/ Cerebral Palsy Be Able To Find A Wife Or GF?

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My old buddy from high school (now in his early 50's) has cerebral palsy. Let's call him "Romeo".

I spoke to him on the phone a few night ago. Romeo has never had a wife or even a GF, his trust fund money is running out, and his Mom won't be around much longer. Romeo said his Mom wondered if he could move in with my family after she's gone, and I was too surprised to react and and simply ignored the statement (no, allowing Romeo to move into our house would never happen). I suggested to Romeo that he try and find a wife with whom he can share the rest of his life with, and he replied that he's too broke to join any dating services.

Later, after hanging up and knowing his financial problems... I decided to offer paying for a year in an on-line dating service for Romeo (who looks relatively handsome although he is "pigeon toed", has a trim build, has a driver's license, has the approximate mental capacity of a junior in high school, likes to talk a lot, has always been a gentle and friendly guy, and would be 100% faithful). I have no idea if he could be active in the bedroom, but I would assume that he could be.

A few questions for people who might be familiar with this type of disability:

- What is the likelyhood of Romeo finding a wife or GF?

- What would be the best on-line dating service for Romeo?

Thanks!!!!

Best Regards,
- Brockmann
online dating or bars are the way to go  (+ info)

Why is pain felt in a myocardial infarction?

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I think it would be either because there is not enough oxygen being delivered, or narrowing of the arteries. Can someone help me out with the answer to this question?
1. The heart is working far harder than in healthy times, trying to push though the extreme narrowing or occlusion. It is a muscle, not so different than other muscles in your body.
2. Tissue is dying, because oxygen levels are low. This is painful.  (+ info)

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