1/15. Late massive haemoptyses from bronchopulmonary collaterals in infarcted segments following pulmonary embolism.Massive, recurrent haemoptyses requiring blood transfusions occurred in a patient who had been diagnosed as having pulmonary thromboembolism 3 months earlier. To the authors' knowledge this is the first case report of this kind, in which massive haemoptyses were proved to be caused by large bronchopulmonary collaterals that had developed in the infarcted lung segments affected by embolism. Selective embolization of the collaterals proved to be therapeutic and life saving.- - - - - - - - - - ranking = 1keywords = thromboembolism (Clic here for more details about this article) |
2/15. Acute renal infarction. Clinical characteristics of 17 patients.We analyzed the medical records of patients with an established diagnosis of acute renal infarction to identify predictive parameters of this rare disease. Seventeen patients (8 male) who were admitted to our emergency department between May 1994 and January 1998 were diagnosed by contrast-enhanced computed tomography (CT) as having acute renal infarction (0.007% of all patients). We screened the records of the 17 patients for a history with increased risk for thromboembolism, clinical symptoms, and urine and blood laboratory results known to be associated with acute renal infarction. A history with increased risk for thromboembolism with 1 or more risk factors was found in 14 of 17 patients (82%); risk factors were atrial fibrillation (n = 11), previous embolism (n = 6), mitral stenosis (n = 6), hypertension (n = 9), and ischemic cardiac disease (n = 7). All patients reported persisting pain predominantly from the flank (n = 11), abdomen (n = 4), and lower back (n = 2). On admission, elevated serum lactate dehydrogenase was found in 16 (94%) patients, and hematuria was found in 12 (71%) of 17 patients. After 24 hours all patients showed an elevated serum lactate dehydrogenase, and 14 (82%) had a positive test for hematuria. Our findings suggest that in all patients presenting with the triad--high risk of a thromboembolic event, persisting flank/abdominal/lower back pain, elevated serum levels of lactate dehydrogenase and/or hematuria within 24 hours after pain onset--contrast-enhanced CT should be performed as soon as possible to rule out or to prove acute renal infarction.- - - - - - - - - - ranking = 2keywords = thromboembolism (Clic here for more details about this article) |
3/15. lung cavitation in primary antiphospholipid syndrome.Pulmonary complications of primary antiphospholipid syndrome are common and diverse, with thromboembolic events counting as the most frequent manifestation. We present the case of a female patient with a diagnosis of primary antiphospholipid syndrome, pulmonary thromboembolism and infarction followed by lung cavitation.- - - - - - - - - - ranking = 1keywords = thromboembolism (Clic here for more details about this article) |
4/15. Bilateral renal infarction in chronic myelomonocytic leukemia on blast crisis.The major complications of myelodysplastic syndromes are related to cytopenia and evolution to acute myeloid leukemia. Bleeding episodes in MDS, although relatively uncommon, are often related to thrombocytopenia. Bleeding may be exacerbated by platelet dysfunction, which is also found frequently. Furthermore, the major hemostatic problem underlying hyperleukocytosis, as evident in patients with MDS on blast crisis, appears to be hemorrhage rather than thrombosis. Acute thromboembolism, which causes occlusion of blood supply and organ infarction, has rarely been observed in patients with MDS. Recently, we encountered an elderly female patient, who had chronic myelomonocytic leukemia with marked myelodysplasia, terminating in blast crisis and bilateral renal infarction. This complication rapidly led to oliguric acute renal failure and mortality.- - - - - - - - - - ranking = 1keywords = thromboembolism (Clic here for more details about this article) |
5/15. diagnosis of liver infarction postpartum.BACKGROUND: patients with antiphospholipid syndrome (APS) have increased risks of developing thromboembolism, and the risk maybe amplified by the hypercoagulable state associated with pregnancy. CASE: A patient presented with severe chest pain, mild pyrexia associated with elevated serum transaminases, and marked neutrophilia after vaginal delivery. liver infarction was diagnosed by spiral computer tomography and treated successfully with anticoagulation. CONCLUSION: liver infarction is a possible diagnosis in a patient with antiphospholipid syndrome who presents with chest or abdominal pain in the postpartum period.- - - - - - - - - - ranking = 1keywords = thromboembolism (Clic here for more details about this article) |
6/15. Congenital antithrombin iii deficiency causing mesenteric venous infarction: a lesson to remember--a case history.Congenital antithrombin iii deficiency is an uncommon but important cause of venous thrombosis, usually presenting with recurrent or atypical venous thromboembolism. Its importance lies in the fact that early recognition can lead to treatment in the acute stage, which can prevent propagation of the disease, and further prophylactic treatment may reduce the incidence of further episodes. The authors present a case of mesenteric venous infarction associated with congenital antithrombin iii deficiency and briefly discuss the diagnosis, surgical implications, and treatment of this disorder.- - - - - - - - - - ranking = 1keywords = thromboembolism (Clic here for more details about this article) |
7/15. Postoperative primary adrenal failure in a patient with anticardiolipin antibodies.A 50-year-old woman with no history of thrombosis or recurrent abortions developed pulmonary thromboembolism and bilateral hemorrhagic adrenal infarction with adrenal failure after hysterectomy for uterine fibroids. Anticardiolipin antibodies (aCL) were detected in high titer and have persisted. She remains well, without further thromboses, taking steroid replacement and warfarin anticoagulation. The initial presentation of aCL related disease can be as thrombotic postoperative complications in middle-age.- - - - - - - - - - ranking = 1keywords = thromboembolism (Clic here for more details about this article) |
8/15. Branch retinal artery occlusion in the churg-strauss syndrome.amaurosis fugax followed by retinal infarction occurred as an early manifestation of exacerbation in a 46-year-old woman with allergic angiitis and granulomatosis (churg-strauss syndrome). There was evidence of widespread intraluminal branch retinal artery occlusions without accompanying retinal vasculitis. Low molecular weight dextran and heparin, in addition to systemic steroid therapy, appeared to be beneficial. A review of the ophthalmic manifestations of the churg-strauss syndrome and of other hypereosinophilic conditions suggests that thromboembolism resulting from a hypercoagulable state may be as important as vasculitis in causing visual symptoms. Specific therapy attempting to alter blood coagulation and rheology may have an important role in these conditions.- - - - - - - - - - ranking = 1keywords = thromboembolism (Clic here for more details about this article) |
9/15. Renal infarction.A 43-year-old woman complaining of left flank pain was found to have renal infarction. New-onset atrial fibrillation suggested thromboembolism, which was confirmed by retrograde urogram and intravenous pyelogram. The patient was treated with heparin and was discharged on coumadin after evaluation of her cardiac disease.- - - - - - - - - - ranking = 1keywords = thromboembolism (Clic here for more details about this article) |
10/15. Neonatal pulmonary infarction. A cause of 'cystlike' lucencies on the chest roentgenogram.Perinatal pulmonary infarction is a difficult and infrequently made diagnosis. A male newborn had infarction of most of the right lung secondary to perinatal pulmonary thromboembolism (PTE). Serial chest roentgenograms initially showed opacity of most of the right lung, followed by the appearance of well-defined radiolucencies resembling pneumatoceles associated with mass effect. By the 25th day of life the localized "cystlike" lucencies were no longer evident and the right lung appeared predominantly hyperlucent. Although the presence of underlying disease makes the diagnosis of PTE in the infant and young child difficult, the clinical features and methods of diagnosis of PTE in this age group are similar to those for the adult. The diagnosis of PTE should be considered in the infant with respiratory distress and unusual findings on chest roentgenography.- - - - - - - - - - ranking = 1keywords = thromboembolism (Clic here for more details about this article) |
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