Cases reported "Horner Syndrome"

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21/32. nasal obstruction after cervical sympathectomy: Horner's syndrome revisited.

    Horner's syndrome (cervical sympathetic paralysis) typically includes ptosis, miosis, enophthalmos and facial anhidrosis. However, the effect of sympathetic denervation upon the nasal mucosa should be included as a prominent feature, since nasal obstruction is often the most disabling sequel. The anatomical, physiological, and historical basis is discussed and surgical management suggested.
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keywords = miosis
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22/32. cluster headache: further observations on the dissociation of pain and autonomic findings.

    A 51-year-old male cluster headache patient had during five bouts in the course of 11 years always had the headache attacks on the left side. Autonomic abnormalities were, however, present on the right side. Pupillometrically, there was thus a Horner-like syndrome on the right (non-symptomatic) side, with miosis and a relatively more marked dilatation of that eye subsequent to topical application of a directly working sympathomimetic agent (phenylephrine) than after an indirectly working one (hydroxyamphetamine), whereas this was not the case on the symptomatic side. The findings on evaporimetry were not as clear-cut as the pupillometric findings; however, even facial sweating was consistent with a pathologic condition on the right (non-symptomatic) side. A primary dichotomy of pain and autonomic signs (that is, not due to change of side of pain localization) thus seems to be present in this case.
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keywords = miosis
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23/32. Congenital Horner's syndrome in otolaryngologic practice.

    It is well known that Horner's syndrome is characterized by ptosis, miosis, and facial anhydrosis. However, the congenital form of Horner's syndrome is relatively uncommon, and it is scarcely known among otolaryngologists. In this report, two cases of congenital Horner's syndrome are described, both encountered recently in the recent author's practice. In the first case, the patient was seeking unnecessary medical treatment for a facial abnormality, not for his eyes. thermography performed on this patient confirmed his complaint. Having had the first experience made diagnosis in the second case easier. Thus, although rare, this syndrome may be encountered in an otolaryngologist's daily practice and it is important to recognize its symptoms. It is suggested that some suffering from this disease may go undetected due to physicians' lack of familiarity with it.
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keywords = miosis
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24/32. Iatrogenic Horner's syndrome secondary to chest tube thoracostomy.

    We report a 22-year-old male who underwent a left thoracostomy and left lower lobectomy for a shotgun wound of the left anterior chest. During surgery, two chest tubes were placed in the left hemithorax at the level of the first and second intervertebral spaces. On the 10th postoperative day, the patient developed a left-sided miosis and ptosis. The diagnosis of a left preganglionic Horner's syndrome was confirmed by pharmacologic testing. The Horner's syndrome was unchanged 3 months after discharge. We conclude that the chest tubes damaged the preganglionic sympathetic fibers of the left orbit resulting in this patient's Horner's syndrome.
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ranking = 1
keywords = miosis
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25/32. horner syndrome during lumbar epidural analgesia for obstetrics.

    horner syndrome (ptosis, miosis, anhidrosis, and facial and conjunctival vasodilation) is a recognized complication of lumbar epidural analgesia for labor and delivery. Alone, it presents no significant risk to mother or fetus, as resolution is spontaneous and complete. horner syndrome may, however, be associated with significant maternal hypotension and therefore should be an indication for close maternal and fetal monitoring to provide reassurance.
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ranking = 1
keywords = miosis
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26/32. Raeder's syndrome associated with internal carotid artery dilation and sinusitis.

    Raeder's syndrome consists of ipsilateral ptosis, miosis and facial pain with intact facial sweating. When not associated with other neurologic signs, the clinical course of this conditions is self-limited. patients will have resolution of facial pain but persistence of miosis and ptosis. Treatment is symptomatic with arteriography reserved for those patients with protracted symptoms or atypical presentations. A case of Raeder's paratrigeminal syndrome is presented with abnormal dilation of the subcavernous portion of the internal carotid artery thought to be secondary to inflammation of the adjacent sphenoid sinus. facial pain and the abnormal dilation of the carotid artery resolved, but miosis and ptosis persisted. Because of the therapeutic indication and prognostic value, an awareness of Raeder's syndrome is stressed when evaluating patients with facial pain or possible Horner's syndrome.
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keywords = miosis
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27/32. Raeder's paratrigeminal syndrome due to spontaneous dissection of the cervical and petrous internal carotid artery.

    The combination of pain, ipsilateral oculosympathetic defect (ptosis and miosis), and ipsilateral trigeminal dysfunction constitutes Raeder's syndrome. We describe a patient with an acute presentation of Raeder's syndrome due to spontaneous internal carotid artery dissection. True trigeminal dysfunction due to carotid dissection is rare, and the potential mechanisms for its involvement are reviewed in this paper. Finally, we remind clinicians to consider dissection in the differential diagnosis of Raeder's syndrome because of its potential for ischemic cerebral neurologic sequelae and suggest early cranial and neck imaging in the evaluation of such patients.
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ranking = 1
keywords = miosis
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28/32. Raeder's syndrome. Report of two cases.

    Raeder's syndrome constitutes facial pain and ipsilateral stenosis of the palpebral fissure, miosis and enophthalmos. It is divided into two groups. Group I includes cases with parasellar cranial nerve involvement and group II without parasellar cranial nerve involvement. It is often difficult to distinguish group II of Raeder's syndrome from Horner's syndrome. The latter is painless and is accompanied by anidrosis of the ipsilateral half of the face. In fact Raeder's syndrome may be caused by any lesion affecting the post-ganglionic oculosympathetic fibers distal to the bifurcation of the common carotid artery. Serious lesions of the internal carotid artery (ICA) may be responsible for this syndrome. This paper deals with two cases of group II of Raeder's syndrome. In the first case, a 60-year-old man, the angiogram revealed an aneurysm of the extracranial part of the ICA just below its entrance into the calvarium. In the second case, a 42-year-old man, a dissective aneurysm of the ICA was found. Surgery was not attempted in either of our patients for different reasons. Their symptoms ameliorated quickly with medical treatment.
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ranking = 1
keywords = miosis
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29/32. Raeder's paratrigeminal syndrome: a case report.

    Raeder's paratrigeminal syndrome is an uncommon neurologic disorder characterized by oculosympathetic paralysis, including ptosis and miosis. In the ED, this syndrome can be easily confused with Horner's syndrome, although with Raeder's syndrome trigeninal nerve irritation and preservation of facial sweating are noted. This report reviews a case of a 62-year-old man who presented to the ED with signs and symptoms consistent with Raeder's syndrome. MRI with angiography revealed a carotid artery dissection, and the patient was admitted and anticoagulated. The syndrome has been associated with head trauma, hypertension, vasculitis, migraine headaches, parasellar mass lesions, and internal carotid artery dissections. Hence, treatment of the patient who has Raeder's syndrome is dependent on the specific underlying lesion.
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ranking = 1
keywords = miosis
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30/32. horner syndrome as an isolated manifestation of an intrapetrous internal carotid artery dissection.

    PURPOSE: To describe the atypical localization and unusual clinical manifestation of an internal carotid artery dissection. methods: We examined a 43-year-old woman who had sudden onset of left ptosis and miosis with vague dysesthesia around her eye. RESULTS: Her examination showed an intrapetrous carotid artery dissection. CONCLUSIONS: The intrapetrous segment is an extremely rare localization for an internal carotid artery dissection. The differential diagnosis of acute horner syndrome should include carotid dissection, which is usually accompanied by other neurologic manifestations.
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ranking = 1
keywords = miosis
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